tPA Contributes To Impaired NMDA Cerebrovasodilation After Traumatic Brain Injury Through Activation of JNK MAPK

OBJECTIVE: NMDA induced pial artery dilation (PAD) is reversed to vasoconstriction after fluid percussion brain injury (FPI). Tissue type plasminogen activator (tPA) is upregulated and the tPA antagonist, EEIIMD, prevents impaired NMDA PAD after FPI. Mitogen activated protein kinase (MAPK), a family...

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Hlavní autoři: Armstead, William M., Kiessling, J Willis, Riley, John, Cines, Douglas B., Higazi, Abd Al-Roof
Médium: Artigo
Jazyk:Inglês
Vydáno: 2011
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Article
On-line přístup:https://ncbi.nlm.nih.gov/pmc/articles/PMC3523283/
https://ncbi.nlm.nih.gov/pubmed/21756552
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1179/016164110X12807570509853
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