S-glutathionylation uncouples eNOS and regulates its cellular and vascular function

Endothelial nitric oxide synthase (eNOS) is critical in the regulation of vascular function, and can generate both nitric oxide (NO) and superoxide (O(2)(•−)), which are key mediators of cellular signalling. In the presence of Ca(2+)/calmodulin, eNOS produces NO, endothelial-derived relaxing factor,...

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Detaylı Bibliyografya
Asıl Yazarlar: Chen, Chun-An, Wang, Tse-Yao, Varadharaj, Saradhadevi, Reyes, Levy A., Hemann, Craig, Hassan Talukder, M. A., Chen, Yeong-Renn, Druhan, Lawrence J., Zweier, Jay L.
Materyal Türü: Artigo
Dil:Inglês
Baskı/Yayın Bilgisi: 2010
Konular:
Article
Online Erişim:https://ncbi.nlm.nih.gov/pmc/articles/PMC3370391/
https://ncbi.nlm.nih.gov/pubmed/21179168
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/nature09599
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