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S-glutathionylation uncouples eNOS and regulates its cellular and vascular function
Endothelial nitric oxide synthase (eNOS) is critical in the regulation of vascular function, and can generate both nitric oxide (NO) and superoxide (O(2)(•−)), which are key mediators of cellular signalling. In the presence of Ca(2+)/calmodulin, eNOS produces NO, endothelial-derived relaxing factor,...
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| Main Authors: | , , , , , , , , |
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| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
2010
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3370391/ https://ncbi.nlm.nih.gov/pubmed/21179168 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/nature09599 |
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