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Memory Impairment in Transgenic Alzheimer Mice Requires Cellular Prion Protein
Soluble oligomers of the amyloid-β (Aβ) peptide are thought to play a key role in the pathophysiology of Alzheimer's disease (AD). Recently, we reported that synthetic Aβ oligomers bind to cellular prion protein (PrP(C)) and that this interaction is required for suppression of synaptic plastici...
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| Main Authors: | , , , , , , |
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| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
Society for Neuroscience
2010
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3323924/ https://ncbi.nlm.nih.gov/pubmed/20445063 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1523/JNEUROSCI.0395-10.2010 |
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