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Rapamycin treatment augments both protein ubiquitination and Akt activation in pressure-overloaded rat myocardium
Ubiquitin-mediated protein degradation is necessary for both increased ventricular mass and survival signaling for compensated hypertrophy in pressure-overloaded (PO) myocardium. Another molecular keystone involved in the hypertrophic growth process is the mammalian target of rapamycin (mTOR), which...
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| Main Authors: | , , , , , , , , |
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| 格式: | Artigo |
| 語言: | Inglês |
| 出版: |
American Physiological Society
2011
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| 主題: | |
| 在線閱讀: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3094075/ https://ncbi.nlm.nih.gov/pubmed/21357504 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1152/ajpheart.00545.2010 |
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