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Lack of β3 integrin signaling contributes to calpain-mediated myocardial cell loss in pressure-overloaded myocardium
Although cardiac hypertrophy initially ensues as a compensatory mechanism it often culminates in congestive heart failure. Based on our earlier studies that calpain and β3 integrin play cell death and survival roles, respectively during pressure-overload (PO) hypertrophy, we investigated if the loss...
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| Autors principals: | , , , |
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| Format: | Artigo |
| Idioma: | Inglês |
| Publicat: |
2010
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| Matèries: | |
| Accés en línia: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3319054/ https://ncbi.nlm.nih.gov/pubmed/20224428 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1097/FJC.0b013e3181d9f5d4 |
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