Lack of β3 integrin signaling contributes to calpain-mediated myocardial cell loss in pressure-overloaded myocardium

Although cardiac hypertrophy initially ensues as a compensatory mechanism it often culminates in congestive heart failure. Based on our earlier studies that calpain and β3 integrin play cell death and survival roles, respectively during pressure-overload (PO) hypertrophy, we investigated if the loss...

Full description

Saved in:
Bibliographic Details
Main Authors: Suryakumar, Geetha, Kasiganesan, Harinath, Balasubramanian, Sundaravadivel, Kuppuswamy, Dhandapani
Format: Artigo
Language:Inglês
Published: 2010
Subjects:
Article
Online Access:https://ncbi.nlm.nih.gov/pmc/articles/PMC3319054/
https://ncbi.nlm.nih.gov/pubmed/20224428
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1097/FJC.0b013e3181d9f5d4
Tags: Add Tag
No Tags, Be the first to tag this record!

Similar Items