In vivo administration of calpeptin attenuates calpain activation and cardiomyocyte loss in pressure-overloaded feline myocardium

Calpain activation is linked to the cleavage of several cytoskeletal proteins and could be an important contributor to the loss of cardiomyocytes and contractile dysfunction during cardiac pressure overload (PO). Using a feline right ventricular (RV) PO model, we analyzed calpain activation during t...

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Detalhes bibliográficos
Main Authors: Mani, Santhosh K., Shiraishi, Hirokazu, Balasubramanian, Sundaravadivel, Yamane, Kentaro, Chellaiah, Meenakshi, Cooper, George, Banik, Naren, Zile, Michael R., Kuppuswamy, Dhandapani
Formato: Artigo
Idioma:Inglês
Publicado em: American Physiological Society 2008
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Articles
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC2494745/
https://ncbi.nlm.nih.gov/pubmed/18487434
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1152/ajpheart.00085.2008
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