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Calpain inhibition preserves myocardial structure and function following myocardial infarction

Cardiac pathology, such as myocardial infarction (MI), activates intracellular proteases that often trigger programmed cell death and contribute to maladaptive changes in myocardial structure and function. To test whether inhibition of calpain, a Ca(2+)-dependent cysteine protease, would prevent the...

תיאור מלא

שמור ב:
מידע ביבליוגרפי
Main Authors: Mani, Santhosh K., Balasubramanian, Sundaravadivel, Zavadzkas, Juozas A., Jeffords, Laura B., Rivers, William T., Zile, Michael R., Mukherjee, Rupak, Spinale, Francis G., Kuppuswamy, Dhandapani
פורמט: Artigo
שפה:Inglês
יצא לאור: American Physiological Society 2009
נושאים:
גישה מקוונת:https://ncbi.nlm.nih.gov/pmc/articles/PMC2781387/
https://ncbi.nlm.nih.gov/pubmed/19734364
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1152/ajpheart.00338.2009
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