Calpain inhibition preserves myocardial structure and function following myocardial infarction

Cardiac pathology, such as myocardial infarction (MI), activates intracellular proteases that often trigger programmed cell death and contribute to maladaptive changes in myocardial structure and function. To test whether inhibition of calpain, a Ca(2+)-dependent cysteine protease, would prevent the...

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Main Authors: Mani, Santhosh K., Balasubramanian, Sundaravadivel, Zavadzkas, Juozas A., Jeffords, Laura B., Rivers, William T., Zile, Michael R., Mukherjee, Rupak, Spinale, Francis G., Kuppuswamy, Dhandapani
格式: Artigo
語言:Inglês
出版: American Physiological Society 2009
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在線閱讀:https://ncbi.nlm.nih.gov/pmc/articles/PMC2781387/
https://ncbi.nlm.nih.gov/pubmed/19734364
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1152/ajpheart.00338.2009
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