Calpain inhibition preserves myocardial structure and function following myocardial infarction

Cardiac pathology, such as myocardial infarction (MI), activates intracellular proteases that often trigger programmed cell death and contribute to maladaptive changes in myocardial structure and function. To test whether inhibition of calpain, a Ca(2+)-dependent cysteine protease, would prevent the...

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Autors principals: Mani, Santhosh K., Balasubramanian, Sundaravadivel, Zavadzkas, Juozas A., Jeffords, Laura B., Rivers, William T., Zile, Michael R., Mukherjee, Rupak, Spinale, Francis G., Kuppuswamy, Dhandapani
Format: Artigo
Idioma:Inglês
Publicat: American Physiological Society 2009
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Articles
Accés en línia:https://ncbi.nlm.nih.gov/pmc/articles/PMC2781387/
https://ncbi.nlm.nih.gov/pubmed/19734364
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1152/ajpheart.00338.2009
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