α-1 Antitrypsin regulates human neutrophil chemotaxis induced by soluble immune complexes and IL-8

Hereditary deficiency of the protein α-1 antitrypsin (AAT) causes a chronic lung disease in humans that is characterized by excessive mobilization of neutrophils into the lung. However, the reason for the increased neutrophil burden has not been fully elucidated. In this study we have demonstrated u...

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Bibliografiske detaljer
Main Authors: Bergin, David A., Reeves, Emer P., Meleady, Paula, Henry, Michael, McElvaney, Oliver J., Carroll, Tomás P., Condron, Claire, Chotirmall, Sanjay H., Clynes, Martin, O’Neill, Shane J., McElvaney, Noel G.
Format: Artigo
Sprog:Inglês
Udgivet: American Society for Clinical Investigation 2010
Fag:
Research Article
Online adgang:https://ncbi.nlm.nih.gov/pmc/articles/PMC2993580/
https://ncbi.nlm.nih.gov/pubmed/21060150
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1172/JCI41196
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