α-1 Antitrypsin regulates human neutrophil chemotaxis induced by soluble immune complexes and IL-8

Hereditary deficiency of the protein α-1 antitrypsin (AAT) causes a chronic lung disease in humans that is characterized by excessive mobilization of neutrophils into the lung. However, the reason for the increased neutrophil burden has not been fully elucidated. In this study we have demonstrated u...

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Bibliografische gegevens
Hoofdauteurs: Bergin, David A., Reeves, Emer P., Meleady, Paula, Henry, Michael, McElvaney, Oliver J., Carroll, Tomás P., Condron, Claire, Chotirmall, Sanjay H., Clynes, Martin, O’Neill, Shane J., McElvaney, Noel G.
Formaat: Artigo
Taal:Inglês
Gepubliceerd in: American Society for Clinical Investigation 2010
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Research Article
Online toegang:https://ncbi.nlm.nih.gov/pmc/articles/PMC2993580/
https://ncbi.nlm.nih.gov/pubmed/21060150
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1172/JCI41196
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