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Effective Post-insult Neuroprotection by a Novel Ca(2+)/ Calmodulin-dependent Protein Kinase II (CaMKII) Inhibitor
Ca(2+)/calmodulin (CaM)-dependent protein kinase II (CaMKII) is a major mediator of physiological glutamate signaling involved in higher brain functions. Here, we show CaMKII involvement in pathological glutamate signaling relevant in stroke. The novel inhibitor tatCN21 was neuroprotective even when...
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| 主要な著者: | , , , , |
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| フォーマット: | Artigo |
| 言語: | Inglês |
| 出版事項: |
American Society for Biochemistry and Molecular Biology
2010
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| 主題: | |
| オンライン･アクセス: | https://ncbi.nlm.nih.gov/pmc/articles/PMC2898334/ https://ncbi.nlm.nih.gov/pubmed/20424167 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1074/jbc.M109.088617 |
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