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Nitric Oxide Induces Ca(2+)-independent Activity of the Ca(2+)/Calmodulin-dependent Protein Kinase II (CaMKII)

Both signaling by nitric oxide (NO) and by the Ca(2+)/calmodulin (CaM)-dependent protein kinase II α isoform (CaMKIIα) are implicated in two opposing forms of synaptic plasticity underlying learning and memory, as well as in excitotoxic/ischemic neuronal cell death. For CaMKIIα, these functions spec...

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Detalhes bibliográficos
Main Authors: Coultrap, Steven J., Bayer, K. Ulrich
Formato: Artigo
Idioma:Inglês
Publicado em: American Society for Biochemistry and Molecular Biology 2014
Assuntos:
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC4094056/
https://ncbi.nlm.nih.gov/pubmed/24855644
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1074/jbc.M114.558254
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