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Nitric Oxide Induces Ca(2+)-independent Activity of the Ca(2+)/Calmodulin-dependent Protein Kinase II (CaMKII)
Both signaling by nitric oxide (NO) and by the Ca(2+)/calmodulin (CaM)-dependent protein kinase II α isoform (CaMKIIα) are implicated in two opposing forms of synaptic plasticity underlying learning and memory, as well as in excitotoxic/ischemic neuronal cell death. For CaMKIIα, these functions spec...
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| Autores principales: | , |
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| Formato: | Artigo |
| Lenguaje: | Inglês |
| Publicado: |
American Society for Biochemistry and Molecular Biology
2014
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| Materias: | |
| Acceso en línea: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4094056/ https://ncbi.nlm.nih.gov/pubmed/24855644 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1074/jbc.M114.558254 |
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