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CaMKII in cerebral ischemia
Ischemic insults on neurons trigger excessive, pathological glutamate release that causes Ca(2+) overload resulting in neuronal cell death (excitotoxicity). The Ca(2+)/calmodulin (CaM)-dependent protein kinase II (CaMKII) is a major mediator of physiological excitatory glutamate signals underlying n...
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| Huvudupphovsmän: | , , , , |
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| Materialtyp: | Artigo |
| Språk: | Inglês |
| Publicerad: |
Nature Publishing Group
2011
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| Ämnen: | |
| Länkar: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3846291/ https://ncbi.nlm.nih.gov/pubmed/21685929 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/aps.2011.68 |
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