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Excision of titin’s cardiac PEVK spring element abolishes PKCα-induced increases in myocardial stiffness
Protein Kinase C-alpha (PKCα) was recently reported to increase myocardial stiffness, an effect that was proposed to be due to phosphorylation of two highly conserved sites (S11878 and S12022) within the proline-gluatamic acid-valine-lysine (PEVK) rich spring element of titin. To test this proposal...
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| Autors principals: | , , , |
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| Format: | Artigo |
| Idioma: | Inglês |
| Publicat: |
2009
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| Matèries: | |
| Accés en línia: | https://ncbi.nlm.nih.gov/pmc/articles/PMC2854275/ https://ncbi.nlm.nih.gov/pubmed/20026128 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.yjmcc.2009.12.006 |
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