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Synthetic amyloid-β oligomers impair long-term memory independently of cellular prion protein
Inability to form new memories is an early clinical sign of Alzheimer’s disease (AD). There is ample evidence that the amyloid-β (Aβ) peptide plays a key role in the pathogenesis of this disorder. Soluble, bio-derived oligomers of Aβ are proposed as the key mediators of synaptic and cognitive dysfun...
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| Autors principals: | , , , , , , , , , , , , , |
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| Format: | Artigo |
| Idioma: | Inglês |
| Publicat: |
National Academy of Sciences
2010
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| Matèries: | |
| Accés en línia: | https://ncbi.nlm.nih.gov/pmc/articles/PMC2836680/ https://ncbi.nlm.nih.gov/pubmed/20133875 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1073/pnas.0911829107 |
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