Age-related loss of phospholipid asymmetry in APP(NLh)/APP(NLh) × PS-1(P264L)/PS-1(P264L) human double knock-in mice: Relevance to Alzheimer disease

Míreanna Comhchosúla

• Proteomics Identification of Specifically Carbonylated Brain Proteins in APP(NLh)/APP(NLh) x PS-1(P264L)/PS-1(P264L) Human Double Mutant Knock-in Mice Model of Alzheimer Disease as a Function of Age
  le: Sultana, Rukhsana, et al.
  Foilsithe: (2011)
• β-Amyloid Mediated Nitration of Manganese Superoxide Dismutase : Implication for Oxidative Stress in a APP(NLh/NLh) X PS-1(P264L/P264L) Double Knock-In Mouse Model of Alzheimer’s Disease
  le: Anantharaman, Muthuswamy, et al.
  Foilsithe: (2006)
• Oxidative damage in brain from human mutant APP/PS-1 double knock-in mice as a function of age
  le: Abdul, Hafiz Mohmmad, et al.
  Foilsithe: (2008)
• Effects of short-term Western diet on cerebral oxidative stress and diabetes related factors in APP × PS1 knock-in mice
  le: Studzinski, Christa M., et al.
  Foilsithe: (2008)
• Proteomics Analysis of Brain Proteins in APP/PS-1 Human Double Mutant Knock-in Mice with Increasing Amyloid β-Peptide Deposition: Insights into the Effects of in vivo Treatment with N-Acetylcysteine as a Potential Therapeutic Intervention in Mild Cognitive Impairment and Alzheimer Disease
  le: Robinson, Renã A. S., et al.
  Foilsithe: (2011)