Oxidative damage in brain from human mutant APP/PS-1 double knock-in mice as a function of age

Lignende værker

• Proteomics Identification of Specifically Carbonylated Brain Proteins in APP(NLh)/APP(NLh) x PS-1(P264L)/PS-1(P264L) Human Double Mutant Knock-in Mice Model of Alzheimer Disease as a Function of Age
  af: Sultana, Rukhsana, et al.
  Udgivet: (2011)
• Age-related loss of phospholipid asymmetry in APP(NLh)/APP(NLh) × PS-1(P264L)/PS-1(P264L) human double knock-in mice: Relevance to Alzheimer disease
  af: Bader Lange, Miranda L., et al.
  Udgivet: (2010)
• Proteomics Analysis of Brain Proteins in APP/PS-1 Human Double Mutant Knock-in Mice with Increasing Amyloid β-Peptide Deposition: Insights into the Effects of in vivo Treatment with N-Acetylcysteine as a Potential Therapeutic Intervention in Mild Cognitive Impairment and Alzheimer Disease
  af: Robinson, Renã A. S., et al.
  Udgivet: (2011)
• β-Amyloid Mediated Nitration of Manganese Superoxide Dismutase : Implication for Oxidative Stress in a APP(NLh/NLh) X PS-1(P264L/P264L) Double Knock-In Mouse Model of Alzheimer’s Disease
  af: Anantharaman, Muthuswamy, et al.
  Udgivet: (2006)
• In vivo Administration of D609 Leads to Protection of Subsequently Isolated Gerbil Brain Mitochondria Subjected to In vitro Oxidative Stress Induced by Amyloid Beta-peptide and Other Oxidative Stressors: Relevance to Alzheimer’s Disease and Other Oxidative Stress-Related Neurodegenerative Disorders.
  af: Ansari, Mubeen Ahmad, et al.
  Udgivet: (2006)