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Alzheimer’s-associated Aβ oligomers show altered structure, immunoreactivity and synaptotoxicity with low doses of oleocanthal
It now appears likely that soluble oligomers of amyloid-β(1-42) peptide, rather than insoluble fibrils, act as the primary neurotoxin in Alzheimer’s disease (AD). Consequently, compounds capable of altering the assembly state of these oligomers (referred to as ADDLs) may have potential for AD therap...
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| 主要な著者: | , , , , , , , |
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| フォーマット: | Artigo |
| 言語: | Inglês |
| 出版事項: |
2009
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| 主題: | |
| オンライン･アクセス: | https://ncbi.nlm.nih.gov/pmc/articles/PMC2808212/ https://ncbi.nlm.nih.gov/pubmed/19631677 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.taap.2009.07.018 |
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