טוען...
PKC phosphorylation of titin's PEVK element-- a novel and conserved pathway for modulating myocardial stiffness
RATIONALE: Protein Kinase C (PKC) regulates contractility of cardiac muscle cells by phosphorylating thin- and thick- filament based proteins. Myocardial sarcomeres also contain a third myofilament, titin, and it is unknown whether titin can be phosphorylated by PKC and if it affects passive tension...
שמור ב:
| Main Authors: | , , , , , , , |
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| פורמט: | Artigo |
| שפה: | Inglês |
| יצא לאור: |
2009
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| נושאים: | |
| גישה מקוונת: | https://ncbi.nlm.nih.gov/pmc/articles/PMC2764991/ https://ncbi.nlm.nih.gov/pubmed/19679839 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1161/CIRCRESAHA.109.198465 |
| תגים: |
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