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A JAK1/JAK2 chimera can sustain alpha and gamma interferon responses.

Cell lines that are mutated in interferon (IFN) responses have been critical in establishing an essential role for the JAK family of nonreceptor tyrosine kinases in interferon signalling. Mutant gamma1A cells have previously been shown to be complemented by overexpression of JAK2. Here, it is shown...

詳細記述

保存先:
書誌詳細
主要な著者: Kohlhuber, F, Rogers, N C, Watling, D, Feng, J, Guschin, D, Briscoe, J, Witthuhn, B A, Kotenko, S V, Pestka, S, Stark, G R, Ihle, J N, Kerr, I M
フォーマット: Artigo
言語:Inglês
出版事項: 1997
主題:
オンライン・アクセス:https://ncbi.nlm.nih.gov/pmc/articles/PMC231795/
https://ncbi.nlm.nih.gov/pubmed/9001223
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