Ca(2+) signalling and PKCα activate increased endothelial permeability by disassembly of VE—cadherin junctions

1. The role of intracellular Ca(2+) mobilization in the mechanism of increased endothelial permeability was studied. Human umbilical vein endothelial cells (HUVECs) were exposed to thapsigargin or thrombin at concentrations that resulted in similar increases in intracellular Ca(2+) concentration ([C...

Deskribapen osoa

Gorde:
Xehetasun bibliografikoak
Egile Nagusiak: Sandoval, Raudel, Malik, Asrar B, Minshall, Richard D, Kouklis, Panos, Ellis, Chad A, Tiruppathi, Chinnaswamy
Formatua: Artigo
Hizkuntza:Inglês
Argitaratua: Blackwell Science Inc 2001
Gaiak:
Original Articles
Sarrera elektronikoa:https://ncbi.nlm.nih.gov/pmc/articles/PMC2278647/
https://ncbi.nlm.nih.gov/pubmed/11389203
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1111/j.1469-7793.2001.0433a.x
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