Ca(2+) signalling and PKCα activate increased endothelial permeability by disassembly of VE—cadherin junctions

1. The role of intracellular Ca(2+) mobilization in the mechanism of increased endothelial permeability was studied. Human umbilical vein endothelial cells (HUVECs) were exposed to thapsigargin or thrombin at concentrations that resulted in similar increases in intracellular Ca(2+) concentration ([C...

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Bibliografiske detaljer
Main Authors: Sandoval, Raudel, Malik, Asrar B, Minshall, Richard D, Kouklis, Panos, Ellis, Chad A, Tiruppathi, Chinnaswamy
Format: Artigo
Sprog:Inglês
Udgivet: Blackwell Science Inc 2001
Fag:
Original Articles
Online adgang:https://ncbi.nlm.nih.gov/pmc/articles/PMC2278647/
https://ncbi.nlm.nih.gov/pubmed/11389203
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1111/j.1469-7793.2001.0433a.x
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