Ca(2+) signalling and PKCα activate increased endothelial permeability by disassembly of VE—cadherin junctions

1. The role of intracellular Ca(2+) mobilization in the mechanism of increased endothelial permeability was studied. Human umbilical vein endothelial cells (HUVECs) were exposed to thapsigargin or thrombin at concentrations that resulted in similar increases in intracellular Ca(2+) concentration ([C...

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Detalhes bibliográficos
Main Authors: Sandoval, Raudel, Malik, Asrar B, Minshall, Richard D, Kouklis, Panos, Ellis, Chad A, Tiruppathi, Chinnaswamy
Formato: Artigo
Idioma:Inglês
Publicado em: Blackwell Science Inc 2001
Assuntos:
Original Articles
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC2278647/
https://ncbi.nlm.nih.gov/pubmed/11389203
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1111/j.1469-7793.2001.0433a.x
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