Ca(2+) signalling and PKCα activate increased endothelial permeability by disassembly of VE—cadherin junctions

1. The role of intracellular Ca(2+) mobilization in the mechanism of increased endothelial permeability was studied. Human umbilical vein endothelial cells (HUVECs) were exposed to thapsigargin or thrombin at concentrations that resulted in similar increases in intracellular Ca(2+) concentration ([C...

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Bibliografski detalji
Glavni autori: Sandoval, Raudel, Malik, Asrar B, Minshall, Richard D, Kouklis, Panos, Ellis, Chad A, Tiruppathi, Chinnaswamy
Format: Artigo
Jezik:Inglês
Izdano: Blackwell Science Inc 2001
Teme:
Original Articles
Online pristup:https://ncbi.nlm.nih.gov/pmc/articles/PMC2278647/
https://ncbi.nlm.nih.gov/pubmed/11389203
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1111/j.1469-7793.2001.0433a.x
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