Ca(2+) signalling and PKCα activate increased endothelial permeability by disassembly of VE—cadherin junctions

1. The role of intracellular Ca(2+) mobilization in the mechanism of increased endothelial permeability was studied. Human umbilical vein endothelial cells (HUVECs) were exposed to thapsigargin or thrombin at concentrations that resulted in similar increases in intracellular Ca(2+) concentration ([C...

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Detaylı Bibliyografya
Asıl Yazarlar: Sandoval, Raudel, Malik, Asrar B, Minshall, Richard D, Kouklis, Panos, Ellis, Chad A, Tiruppathi, Chinnaswamy
Materyal Türü: Artigo
Dil:Inglês
Baskı/Yayın Bilgisi: Blackwell Science Inc 2001
Konular:
Original Articles
Online Erişim:https://ncbi.nlm.nih.gov/pmc/articles/PMC2278647/
https://ncbi.nlm.nih.gov/pubmed/11389203
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1111/j.1469-7793.2001.0433a.x
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