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Mutagenicity of 5-aza-2′-deoxycytidine is mediated by the mammalian DNA methyltransferase

The cytosine analog 5-aza-2′-deoxycytidine has been used clinically to reactivate genes silenced by DNA methylation. In particular, patients with β-thalassemia show fetal globin expression after administration of this hypomethylating drug. In addition, silencing of tumor suppressor gene expression b...

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Auteurs principaux: Jackson-Grusby, Laurie, Laird, Peter W., Magge, Suresh N., Moeller, Benjamin J., Jaenisch, Rudolf
Format: Artigo
Langue:Inglês
Publié: The National Academy of Sciences of the USA 1997
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Accès en ligne:https://ncbi.nlm.nih.gov/pmc/articles/PMC20784/
https://ncbi.nlm.nih.gov/pubmed/9114051
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