An oncogenic form of p53 confers a dominant, gain-of-function phenotype that disrupts spindle checkpoint control

Lignende værker

• Characterization of the p53-Dependent Postmitotic Checkpoint following Spindle Disruption
  af: Lanni, Jennifer S., et al.
  Udgivet: (1998)
• Regulators of Oncogenic Mutant TP53 Gain of Function
  af: Yamamoto, Satomi, et al.
  Udgivet: (2018)
• FOXM1 allows human keratinocytes to bypass the oncogene-induced differentiation checkpoint in response to gain of MYC or loss of p53
  af: Molinuevo, R, et al.
  Udgivet: (2017)
• Spindle assembly checkpoint and p53 deficiencies cooperate for tumorigenesis in mice
  af: Chi, Ya-Hui, et al.
  Udgivet: (2009)
• The kinetochore encodes a mechanical switch to disrupt spindle assembly checkpoint signaling
  af: Aravamudhan, Pavithra, et al.
  Udgivet: (2015)