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Priming and effector dependence on insulin B:9–23 peptide in NOD islet autoimmunity
NOD mice with knockout of both native insulin genes and a mutated proinsulin transgene, alanine at position B16 in preproinsulin (B16:A-dKO mice), do not develop diabetes. Transplantation of NOD islets, but not bone marrow, expressing native insulin sequences (tyrosine at position B16) into B16:A-dK...
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| Autors principals: | , , , , , , , , , , |
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| Format: | Artigo |
| Idioma: | Inglês |
| Publicat: |
American Society for Clinical Investigation
2007
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| Matèries: | |
| Accés en línia: | https://ncbi.nlm.nih.gov/pmc/articles/PMC1904318/ https://ncbi.nlm.nih.gov/pubmed/17607359 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1172/JCI31368 |
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