c-Fms and the α(v)β(3) integrin collaborate during osteoclast differentiation
β(3) integrin–null osteoclasts are dysfunctional, but their numbers are increased in vivo. In vitro, however, the number of β(3)(–/–) osteoclasts is reduced because of arrested differentiation. This paradox suggests cytokine regulation of β(3)(–/–) osteoclastogenesis differs in vitro and in vivo. In...
Tallennettuna:
| Julkaisussa: | J Clin Invest |
|---|---|
| Päätekijät: | , , , , |
| Aineistotyyppi: | Artigo |
| Kieli: | Inglês |
| Julkaistu: |
American Society for Clinical Investigation
2003
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| Aiheet: | |
| Linkit: | https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC151897/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/12618529/ https://ncbi.nlm.nih.govhttps://doi.org/10.1172/JCI16924 |
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