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c-Fms and the α(v)β(3) integrin collaborate during osteoclast differentiation
β(3) integrin–null osteoclasts are dysfunctional, but their numbers are increased in vivo. In vitro, however, the number of β(3)(–/–) osteoclasts is reduced because of arrested differentiation. This paradox suggests cytokine regulation of β(3)(–/–) osteoclastogenesis differs in vitro and in vivo. In...
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| Asıl Yazarlar: | , , , , |
|---|---|
| Materyal Türü: | Artigo |
| Dil: | Inglês |
| Baskı/Yayın Bilgisi: |
American Society for Clinical Investigation
2003
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| Konular: | |
| Online Erişim: | https://ncbi.nlm.nih.gov/pmc/articles/PMC151897/ https://ncbi.nlm.nih.gov/pubmed/12618529 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1172/JCI200316924 |
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