c-Fms and the α(v)β(3) integrin collaborate during osteoclast differentiation

β(3) integrin–null osteoclasts are dysfunctional, but their numbers are increased in vivo. In vitro, however, the number of β(3)(–/–) osteoclasts is reduced because of arrested differentiation. This paradox suggests cytokine regulation of β(3)(–/–) osteoclastogenesis differs in vitro and in vivo. In...

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Pubblicato in:	J Clin Invest
Autori principali:	Faccio, Roberta, Takeshita, Sunao, Zallone, Alberta, Ross, F. Patrick, Teitelbaum, Steven L.
Natura:	Artigo
Lingua:	Inglês
Pubblicazione:	American Society for Clinical Investigation 2003
Soggetti:	Article
Accesso online:	https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC151897/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/12618529/ https://ncbi.nlm.nih.govhttps://doi.org/10.1172/JCI16924
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