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Runaway domain swapping in amyloid-like fibrils of T7 endonuclease I

Amyloid fibrils are associated with >20 fatal human disorders, including Alzheimer's, Parkinson's, and prion diseases. Knowledge of how soluble proteins assemble into amyloid fibrils remains elusive despite its potential usefulness for developing diagnostics and therapeutics. In at leas...

Täydet tiedot

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Bibliografiset tiedot
Päätekijät: Guo, Zhefeng, Eisenberg, David
Aineistotyyppi: Artigo
Kieli:Inglês
Julkaistu: National Academy of Sciences 2006
Aiheet:
Linkit:https://ncbi.nlm.nih.gov/pmc/articles/PMC1472426/
https://ncbi.nlm.nih.gov/pubmed/16698921
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1073/pnas.0602607103
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