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The threshold for polyglutamine-expansion protein aggregation and cellular toxicity is dynamic and influenced by aging in Caenorhabditis elegans

Studies of the mutant gene in Huntington's disease, and for eight related neurodegenerative disorders, have identified polyglutamine (polyQ) expansions as a basis for cellular toxicity. This finding has led to a disease hypothesis that protein aggregation and cellular dysfunction can occur at a...

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Autors principals: Morley, James F., Brignull, Heather R., Weyers, Jill J., Morimoto, Richard I.
Format: Artigo
Idioma:Inglês
Publicat: National Academy of Sciences 2002
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Accés en línia:https://ncbi.nlm.nih.gov/pmc/articles/PMC124929/
https://ncbi.nlm.nih.gov/pubmed/12122205
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1073/pnas.152161099
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