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Natural genetic variation determines susceptibility to aggregation or toxicity in a C. elegans model for polyglutamine disease

BACKGROUND: Monogenic gain-of-function protein aggregation diseases, including Huntington’s disease, exhibit substantial variability in age of onset, penetrance, and clinical symptoms, even between individuals with similar or identical mutations. This difference in phenotypic expression of proteotox...

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Detalhes bibliográficos
Main Authors: Gidalevitz, Tali, Wang, Ning, Deravaj, Tanuja, Alexander-Floyd, Jasmine, Morimoto, Richard I
Formato: Artigo
Idioma:Inglês
Publicado em: BioMed Central 2013
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Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC3816611/
https://ncbi.nlm.nih.gov/pubmed/24079614
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1186/1741-7007-11-100
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