Persistence of Peptide-induced CD4(+) T cell Anergy In Vitro

Por Ryan, Kelli R., Evavold, Brian D.
Publicado em 1998

A unique unresponsive CD4+ T cell phenotype post TCR antagonism

Por Edwards, Lindsay J., Evavold, Brian D.
Publicado em 2009

T cell recognition of weak ligands: roles of signaling, receptor number, and affinity

Por Edwards, Lindsay J., Evavold, Brian D.
Publicado em 2011

Destabilization of peptide: MHC interaction induces IL-2 resistant anergy in diabetogenic T cells

Por Edwards, Lindsay J., Evavold, Brian D.
Publicado no J Autoimmun (2013)

Lower Affinity T Cells are Critical Components and Active Participants of the Immune Response

Por Martinez, Ryan J., Evavold, Brian D.
Publicado no Front Immunol (2015)

MHC Bias by T Cell Receptors: Genetic Evidence for MHC and TCR Co-Evolution

Por Baker, Brian M., Evavold, Brian D.
Publicado no Trends Immunol (2016)

Dissociation of peripheral T cell responses from thymocyte negative selection by weak agonists supports a spare receptor model of T cell activation

Por McNeil, Lisa K., Evavold, Brian D.
Publicado em 2002

Pathogenic MOG-reactive CD8+ T cells require MOG-reactive CD4+ T cells for sustained CNS inflammation during chronic EAE

Por Bettini, Maria, Rosenthal, Kristen, Evavold, Brian D.
Publicado em 2009

Manipulating Antigenic Ligand Strength to Selectively Target Myelin-Reactive CD4+ T Cells in EAE

Por Sabatino, Joseph J., Rosenthal, Kristen M., Evavold, Brian D.
Publicado em 2009

Peptide Immunization Elicits Polyomavirus-Specific MHC Class Ib-Restricted CD8 T Cells in MHC Class Ia Allogeneic Mice

Por Hofstetter, Amelia R., Evavold, Brian D., Lukacher, Aron E.
Publicado em 2013

Monitoring the Dynamics of T Cell Clonal Diversity Using Recombinant Peptide:MHC Technology

Por Blanchfield, J. Lori, Shorter, Shayla K., Evavold, Brian D.
Publicado em 2013

Antigen-specific TCR–pMHC catch bonds trigger signaling by fast accumulation of force-prolonged bond lifetimes

Por Liu, Baoyu, Chen, Wei, Evavold, Brian D., Zhu, Cheng
Publicado em 2014

Progression of relapsing-remitting demyelinating disease does not require increased TCR affinity or epitope spread

Por Kersh, Anna E., Edwards, Lindsay J., Evavold, Brian D.
Publicado em 2014

Identification of T cell clones without the need for sequencing

Por Martinez, Ryan J., Neeld, Dennis K., Evavold, Brian D.
Publicado no J Immunol Methods (2015)

High prevalence of low affinity peptide–MHC II tetramer–negative effectors during polyclonal CD4(+) T cell responses

Por Sabatino, Joseph J., Huang, Jun, Zhu, Cheng, Evavold, Brian D.
Publicado em 2011

NFM cross-reactivity to MOG does not expand a critical threshold level of high affinity T cells necessary for onset of demyelinating disease

Por Blanchfield, Lori, Sabatino, Joseph J., Lawrence, Laurel, Evavold, Brian D.
Publicado no J Immunol (2017)

Regulatory and T effector cells have overlapping low to high ranges in TCR affinities for self during demyelinating disease

Por Hood, Jennifer D., Zarnitsyna, Veronika I., Zhu, Cheng, Evavold, Brian D.
Publicado no J Immunol (2015)

Low-affinity CD4+ T cells are major responders in the primary immune response

Por Martinez, Ryan J., Andargachew, Rakieb, Martinez, Hunter A., Evavold, Brian D.
Publicado no Nat Commun (2016)

Insights from in situ analysis of TCR–pMHC recognition: response of an interaction network

Por Zhu, Cheng, Jiang, Ning, Huang, Jun, Zarnitsyna, Veronika I., Evavold, Brian D.
Publicado em 2013

Accumulation of serial forces on TCR and CD8 frequently applied by agonist pMHC triggers calcium in T cell

Por Pryshchep, Sergey, Zarnitsyna, Veronika I., Hong, Jinsung, Evavold, Brian D., Zhu, Cheng
Publicado em 2014