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Inhibition of Herpes Simplex Virus Reactivation by Dipyridamole
Herpes simplex virus (HSV) reactivation from latency was investigated. Reactivation of thymidine kinase-negative HSV, which is defective for reactivation, was greatly enhanced by thymidine (TdR). The reactivation-enhancing effect of TdR was blocked by dipyridamole (DPM), a known nucleoside transport...
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| Main Authors: | , , |
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| Formáid: | Artigo |
| Teanga: | Inglês |
| Foilsithe: |
American Society for Microbiology
2001
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| Ábhair: | |
| Rochtain Ar Líne: | https://ncbi.nlm.nih.gov/pmc/articles/PMC90893/ https://ncbi.nlm.nih.gov/pubmed/11709364 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1128/AAC.45.12.3657-3659.2001 |
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