Inhibition of Herpes Simplex Virus Reactivation by Dipyridamole

Herpes simplex virus (HSV) reactivation from latency was investigated. Reactivation of thymidine kinase-negative HSV, which is defective for reactivation, was greatly enhanced by thymidine (TdR). The reactivation-enhancing effect of TdR was blocked by dipyridamole (DPM), a known nucleoside transport...

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Detaylı Bibliyografya
Asıl Yazarlar: Tenser, Richard B., Gaydos, Andrew, Hay, Kathleen A.
Materyal Türü: Artigo
Dil:Inglês
Baskı/Yayın Bilgisi: American Society for Microbiology 2001
Konular:
Antiviral Agents
Online Erişim:https://ncbi.nlm.nih.gov/pmc/articles/PMC90893/
https://ncbi.nlm.nih.gov/pubmed/11709364
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1128/AAC.45.12.3657-3659.2001
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