Inhibition of Herpes Simplex Virus Reactivation by Dipyridamole
Herpes simplex virus (HSV) reactivation from latency was investigated. Reactivation of thymidine kinase-negative HSV, which is defective for reactivation, was greatly enhanced by thymidine (TdR). The reactivation-enhancing effect of TdR was blocked by dipyridamole (DPM), a known nucleoside transport...
Uloženo v:
| Vydáno v: | Antimicrob Agents Chemother |
|---|---|
| Hlavní autoři: | , , |
| Médium: | Artigo |
| Jazyk: | Inglês |
| Vydáno: |
American Society for Microbiology (ASM)
2001
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| Témata: | |
| On-line přístup: | https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC90893/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/11709364/ https://ncbi.nlm.nih.govhttps://doi.org/10.1128/AAC.45.12.3657-3659.2001 |
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