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Inhibition of Herpes Simplex Virus Reactivation by Dipyridamole

Herpes simplex virus (HSV) reactivation from latency was investigated. Reactivation of thymidine kinase-negative HSV, which is defective for reactivation, was greatly enhanced by thymidine (TdR). The reactivation-enhancing effect of TdR was blocked by dipyridamole (DPM), a known nucleoside transport...

Πλήρης περιγραφή

Αποθηκεύτηκε σε:
Λεπτομέρειες βιβλιογραφικής εγγραφής
Εκδόθηκε σε:Antimicrob Agents Chemother
Κύριοι συγγραφείς: Tenser, Richard B., Gaydos, Andrew, Hay, Kathleen A.
Μορφή: Artigo
Γλώσσα:Inglês
Έκδοση: American Society for Microbiology (ASM) 2001
Θέματα:
Διαθέσιμο Online:https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC90893/
https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/11709364/
https://ncbi.nlm.nih.govhttps://doi.org/10.1128/AAC.45.12.3657-3659.2001
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