Protein-Damaging Stresses Activate c-Jun N-Terminal Kinase via Inhibition of Its Dephosphorylation: a Novel Pathway Controlled by HSP72

Various stresses activate the c-Jun N-terminal kinase (JNK), which is involved in the regulation of many aspects of cellular physiology, including apoptosis. Here we demonstrate that in contrast to UV irradiation, heat shock causes little or no stimulation of the JNK-activating kinase SEK1, while kn...

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Библиографические подробности

Опубликовано в::	Mol Cell Biol
Главные авторы:	Meriin, Anatoli B., Yaglom, Julia A., Gabai, Vladimir L., Mosser, Dick D., Zon, Leonard, Sherman, Michael Y.
Формат:	Artigo
Язык:	Inglês
Опубликовано:	Taylor & Francis 1999
Предметы:	Cell Growth and Development
Online-ссылка:	https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC84047/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/10082520/ https://ncbi.nlm.nih.govhttps://doi.org/10.1128/mcb.19.4.2547
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