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Methylglyoxal induces p53 activation and inhibits mTORC1 in human umbilical vein endothelial cells
Methylglyoxal (MGO), a precursor of advanced glycation end products (AGEs), is regarded as a pivotal mediator of vascular damage in patients with diabetes. We have previously reported that MGO induces transcriptional changes compatible with p53 activation in cultured human endothelial cells. To furt...
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| Publicat a: | Sci Rep |
|---|---|
| Autors principals: | , , , , , , , |
| Format: | Artigo |
| Idioma: | Inglês |
| Publicat: |
Nature Publishing Group UK
2021
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| Matèries: | |
| Accés en línia: | https://ncbi.nlm.nih.gov/pmc/articles/PMC8044125/ https://ncbi.nlm.nih.gov/pubmed/33850227 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/s41598-021-87561-9 |
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