Tumor necrosis factor alpha-mediated inflammation and remodeling of the extracellular matrix underlies aortic stiffening induced by the common chemotherapeutic agent doxorubicin

Lignende værker

• Apigenin restores endothelial function by ameliorating oxidative stress, reverses aortic stiffening, and mitigates vascular inflammation with aging
  af: Clayton, Zachary S., et al.
  Udgivet: (2021)
• Accelerated aging of the brain transcriptome by the common chemotherapeutic doxorubicin
  af: Cavalier, Alyssa N., et al.
  Udgivet: (2021)
• The Gut Microbiome-Derived Metabolite Trimethylamine N-Oxide Induces Aortic Stiffening and Increases Systolic Blood Pressure with Aging in Mice and Humans
  af: Brunt, Vienna E., et al.
  Udgivet: (2021)
• Doxorubicin-Induced Oxidative Stress and Endothelial Dysfunction in Conduit Arteries Is Prevented by Mitochondrial-Specific Antioxidant Treatment
  af: Clayton, Zachary S., et al.
  Udgivet: (2020)
• 2.7 THE GUT-DERIVED METABOLITE TRIMETHYLAMINE N-OXIDE INDUCES LARGE ELASTIC ARTERY STIFFENING AND ENDOTHELIAL DYSFUNCTION IN YOUNG MICE
  af: Vienna Brunt, et al.
  Udgivet: (2017-12-01)