Protease-activated Receptor 4 causes Akt phosphorylation independently of PI3 kinase pathways.

Lignende værker

• Trypsin causes platelet activation independently of known protease-activated receptors
  af: Mao, Yingying, et al.
  Udgivet: (2013)
• Protein kinase C- and calcium-regulated pathways independently synergize with Gi pathways in agonist-induced fibrinogen receptor activation.
  af: Quinton, Todd M, et al.
  Udgivet: (2002)
• G(q)-mediated Akt translocation to the membrane: a novel PIP(3)-independent mechanism in platelets
  af: Badolia, Rachit, et al.
  Udgivet: (2015)
• PDK1 selectively phosphorylates Thr(308) on Akt and contributes to human platelet functional responses
  af: Dangelmaier, Carol, et al.
  Udgivet: (2013)
• Role of Phosphoinositide 3-Kinase β in Glycoprotein VI-mediated Akt Activation in Platelets
  af: Kim, Soochong, et al.
  Udgivet: (2009)