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The tyrosine kinase v‐Src modifies cytotoxicities of anticancer drugs targeting cell division
v‐Src oncogene causes cell transformation through its strong tyrosine kinase activity. We have revealed that v‐Src‐mediated cell transformation occurs at a low frequency and it is attributed to mitotic abnormalities‐mediated chromosome instability. v‐Src directly phosphorylates Tyr‐15 of cyclin‐depe...
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| Pubblicato in: | J Cell Mol Med |
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| Autori principali: | , , , , , , , , |
| Natura: | Artigo |
| Lingua: | Inglês |
| Pubblicazione: |
John Wiley and Sons Inc.
2021
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| Soggetti: | |
| Accesso online: | https://ncbi.nlm.nih.gov/pmc/articles/PMC7875926/ https://ncbi.nlm.nih.gov/pubmed/33465289 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1111/jcmm.16270 |
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