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The tyrosine kinase v‐Src modifies cytotoxicities of anticancer drugs targeting cell division
v‐Src oncogene causes cell transformation through its strong tyrosine kinase activity. We have revealed that v‐Src‐mediated cell transformation occurs at a low frequency and it is attributed to mitotic abnormalities‐mediated chromosome instability. v‐Src directly phosphorylates Tyr‐15 of cyclin‐depe...
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| 出版年: | J Cell Mol Med |
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| 主要な著者: | , , , , , , , , |
| フォーマット: | Artigo |
| 言語: | Inglês |
| 出版事項: |
John Wiley and Sons Inc.
2021
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| 主題: | |
| オンライン･アクセス: | https://ncbi.nlm.nih.gov/pmc/articles/PMC7875926/ https://ncbi.nlm.nih.gov/pubmed/33465289 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1111/jcmm.16270 |
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