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Ca(2+) Release via IP(3) Receptors Shapes the Cardiac Ca(2+) Transient for Hypertrophic Signaling
Calcium (Ca(2+)) plays a central role in mediating both contractile function and hypertrophic signaling in ventricular cardiomyocytes. L-type Ca(2+) channels trigger release of Ca(2+) from ryanodine receptors for cellular contraction, whereas signaling downstream of G-protein-coupled receptors stimu...
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| 出版年: | Biophys J |
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| 主要な著者: | , , , , , , |
| フォーマット: | Artigo |
| 言語: | Inglês |
| 出版事項: |
The Biophysical Society
2020
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| 主題: | |
| オンライン･アクセス: | https://ncbi.nlm.nih.gov/pmc/articles/PMC7499065/ https://ncbi.nlm.nih.gov/pubmed/32871099 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.bpj.2020.08.001 |
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