Restoring Herpesvirus Entry Mediator (HVEM) Immune Function in HVEM(−/−) Mice Rescues Herpes Simplex Virus 1 Latency and Reactivation Independently of Binding to Glycoprotein D

Lignende værker

• Expression of Murine CD80 by Herpes Simplex Virus 1 in Place of Latency-Associated Transcript (LAT) Can Compensate for Latency Reactivation and Anti-apoptotic Functions of LAT
  af: Jaggi, Ujjaldeep, et al.
  Udgivet: (2020)
• The Absence of Lymphotoxin-α, a Herpesvirus Entry Mediator (HVEM) Ligand, Affects Herpes Simplex Virus 1 Infection In Vivo Differently than the Absence of Other HVEM Cellular Ligands
  af: Wang, Shaohui, et al.
  Udgivet: (2019)
• Role of Herpes Simplex Virus Type 1 (HSV-1) Glycoprotein K (gK) Pathogenic CD8(+) T Cells in Exacerbation of Eye Disease
  af: Jaggi, Ujjaldeep, et al.
  Udgivet: (2018)
• CD80 Plays a Critical Role in Increased Inflammatory Responses in Herpes Simplex Virus 1-Infected Mouse Corneas
  af: Tormanen, Kati, et al.
  Udgivet: (2020)
• Interactions between Herpesvirus Entry Mediator (TNFRSF14) and Latency-Associated Transcript during Herpes Simplex Virus 1 Latency
  af: Allen, Sariah J., et al.
  Udgivet: (2014)