The Absence of Lymphotoxin-α, a Herpesvirus Entry Mediator (HVEM) Ligand, Affects Herpes Simplex Virus 1 Infection In Vivo Differently than the Absence of Other HVEM Cellular Ligands

Lignende værker

• Restoring Herpesvirus Entry Mediator (HVEM) Immune Function in HVEM(−/−) Mice Rescues Herpes Simplex Virus 1 Latency and Reactivation Independently of Binding to Glycoprotein D
  af: Tormanen, Kati, et al.
  Udgivet: (2020)
• Herpes Simplex Virus 1 Latency and the Kinetics of Reactivation Are Regulated by a Complex Network of Interactions between the Herpesvirus Entry Mediator, Its Ligands (gD, BTLA, LIGHT, and CD160), and the Latency-Associated Transcript
  af: Wang, Shaohui, et al.
  Udgivet: (2018)
• Roles of Type 1, 2, and 3 Innate Lymphoid Cells in Herpes Simplex Virus 1 Infection In Vitro and In Vivo
  af: Hirose, Satoshi, et al.
  Udgivet: (2019)
• Interactions between Herpesvirus Entry Mediator (TNFRSF14) and Latency-Associated Transcript during Herpes Simplex Virus 1 Latency
  af: Allen, Sariah J., et al.
  Udgivet: (2014)
• CD80 Plays a Critical Role in Increased Inflammatory Responses in Herpes Simplex Virus 1-Infected Mouse Corneas
  af: Tormanen, Kati, et al.
  Udgivet: (2020)