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Rb family-independent activating E2F increases genome stability, promotes Homologous Recombination, and decreases Non-Homologous End Joining
The retinoblastoma protein Rb is a prototype tumor suppressor inactivated in a variety of cancers. In addition to deregulated cell proliferation, Rb inactivation also causes genome instability that contributes to tumorigenesis. Although the genome instability effects of Rb inactivation was shown to...
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| Publicado no: | Mech Dev |
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| Main Authors: | , , , |
| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
2020
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC7295657/ https://ncbi.nlm.nih.gov/pubmed/32217105 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.mod.2020.103607 |
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