Absence of Functional Na(v)1.8 Channels in Non-diseased Atrial and Ventricular Cardiomyocytes

PURPOSE: Several studies have indicated a potential role for SCN10A/Na(V)1.8 in modulating cardiac electrophysiology and arrhythmia susceptibility. However, by which mechanism SCN10A/Na(V)1.8 impacts on cardiac electrical function is still a matter of debate. To address this, we here investigated th...

Täydet tiedot

Tallennettuna:
Bibliografiset tiedot
Julkaisussa:Cardiovasc Drugs Ther
Päätekijät: Casini, Simona, Marchal, Gerard A., Kawasaki, Makiri, Nariswari, Fransisca A., Portero, Vincent, van den Berg, Nicoline W.E., Guan, Kaomei, Driessen, Antoine H.G., Veldkamp, Marieke W., Mengarelli, Isabella, de Groot, Joris R., Verkerk, Arie O., Remme, Carol Ann
Aineistotyyppi: Artigo
Kieli:Inglês
Julkaistu: Springer US 2020
Aiheet:
Original Article
Linkit:https://ncbi.nlm.nih.gov/pmc/articles/PMC6994555/
https://ncbi.nlm.nih.gov/pubmed/31916131
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1007/s10557-019-06925-6
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