GRK6 regulates the hemostatic response to injury through its rate-limiting effects on GPCR signaling in platelets

G protein–coupled receptors (GPCRs) mediate the majority of platelet activation in response to agonists. However, questions remain regarding the mechanisms that provide negative feedback toward activated GPCRs to limit platelet activation and thrombus formation. Here we provide the first evidence th...

詳細記述

保存先:
書誌詳細
出版年:Blood Adv
主要な著者: Chen, Xi, Gupta, Shuchi, Cooper, Matthew, DeHelian, Daniel, Zhao, Xuefei, Naik, Meghna U., Wurtzel, Jeremy G. T., Stalker, Timothy J., Goldfinger, Lawrence E., Benovic, Jeffrey, Brass, Lawrence F., McKenzie, Steven E., Naik, Ulhas P., Ma, Peisong
フォーマット: Artigo
言語:Inglês
出版事項: American Society of Hematology 2020
主題:
Thrombosis and Hemostasis
オンライン･アクセス:https://ncbi.nlm.nih.gov/pmc/articles/PMC6960467/
https://ncbi.nlm.nih.gov/pubmed/31899801
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1182/bloodadvances.2019000467
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