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Smooth muscle cell–specific fibronectin-EDA mediates phenotypic switching and neointimal hyperplasia
Fibronectin–splice variant containing extra domain A (Fn-EDA) is associated with smooth muscle cells (SMCs) following vascular injury. The role of SMC-derived Fn-EDA in SMC phenotypic switching or its implication in neointimal hyperplasia remains unclear. Herein, using human coronary artery sections...
Uloženo v:
| Vydáno v: | J Clin Invest |
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| Hlavní autoři: | , , , , , , , , , |
| Médium: | Artigo |
| Jazyk: | Inglês |
| Vydáno: |
American Society for Clinical Investigation
2019
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| Témata: | |
| On-line přístup: | https://ncbi.nlm.nih.gov/pmc/articles/PMC6934199/ https://ncbi.nlm.nih.gov/pubmed/31763999 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1172/JCI124708 |
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